Arrhythmias Year 4 Internal Medicine Notes OmpathStudy
Year 4 Internal Medicine study notes: Arrhythmias. Full notes on OmpathStudy. Free notes for medical students worldwide.
Arrhythmias Year 4 · Internal Medicine Collection: 4TH YEAR ALL NOTES / IMED / I MED BEAUTIFUL NOTES / CVS -- ● Objectives:\n● Identify the common arrhythmias\n● Know the differential diagnosis of heart rhythm disorder.\n● Learn the treatment modalities and diagnosis of atrial fibrillation.\n● Know the risk stratification of atrial fibrillation patient. [ Color index : Important Notes Extra ]\n[ Editing file Feedback Share your notes Shared notes Twitter ] \n● Resources:\n● 435 slides, 434 & 432 teams\n\n ● Done by: Khawla AlAmmari & Fahad AlAbdullatif & Mana AlMuhaideb\n● Team sub-leader: Mana AlMuhaideb\n● Team leaders: Khawla AlAmmari & Fahad AlAbdullatif\n● Revised by: Ahmed Al Yahya \n\n Introduction to Cardiac Arrhythmias\n\n ● The heartbeat is normally initiated by an electrical discharge from the sinoatrial (sinus) node. The atria and\nventricles then depolarize sequentially as electrical depolarisation passes through specialized conducting\ntissues.The sinus node acts as a pacemaker and its intrinsic rate is regulated by the autonomic nervous system.\n● A reduction of parasympathetic tone or an increase in sympathetic stimulation leads to tachycardia; conversely,\nincreased parasympathetic tone or decreased sympathetic stimulation produces bradycardia. The sinus rate in\nwomen is slightly faster than in men.\n● A cardiac arrhythmia is a disturbance of the electrical rhythm of the heart. Arrhythmias are often a\nmanifestation of structural heart disease but may also occur because of abnormal conduction or depolarization in\nan otherwise healthy heart.\n● An arrhythmia may be ‘supraventricular’ (sinus, atrial or junctional) or ventricular in origin. Supraventricular\nrhythms usually produce narrow QRS complexes because the ventricles are depolarized in their normal sequence\nvia the AV node and bundle of His. In contrast, ventricular rhythms produce broad, bizarre QRS complexes because\nthe ventricles are activated in an abnormal sequence.\n\n ● There are two main types of arrhythmia : (Based on rate)\n1) Bradycardia: the heart rate is s low (<60 b.p.m. during the day or <50 b.p.m. at night).\nSlower heart rates are more likely to cause symptomatic arrhythmias.\n1.\n2) Tachycardia: the heart rate is fast ( 100 b.p.m.). Tachycardias are more likely to be symptomatic\nwhen the arrhythmia is fast and sustained. There are three main mechanisms of tachycardia :\n\nIncreased automaticity (In The tachycardia is produced by repeated spontaneous depolarisation of an ectopic focus, response to any kind of stress) often in response to catecholamines.\n\nRe-entry The tachycardia is initiated by an ectopic beat and sustained by a re-entry circuit . Most\ntachyarrhythmias are due to re-entry.\n\nTriggered activity It is a form of secondary depolarisation arising from an incompletely repolarized cell\nmembrane. \n\n Atrial tachyarrhythmias \nWE ARE 100% SURE THAT YOU WILL UNDERSTAND EVERYTHING ABOUT AF AFTER WATCHING THIS VIDEO 1) Atrial fibrillation (AF) :\n- Atrial fibrillation (AF) is the most common sustained cardiac arrhythmia with an overall prevalence of 0.5%\nin the adult population of the UK.\n➔ Atrial fibrillation accounts for 1/3 of all patient discharges with arrhythmia as principal diagnosis.1\n- The prevalence rises with age, affecting 1% of those aged 60–64 years, increasing to 9% of those aged over 80\nyears.\n- AF is a complex arrhythmia characterised by both :\n1) abnormal automatic firing.\n2) The presence of multiple interacting re-entry circuits looping around the atria.\n➔ AF becomes sustained because of re-entrant conduction within the atria or sometimes because of\ncontinuous ectopic firing.\n- Episodes of atrial fibrillation are initiated by rapid bursts of ectopic beats arising from conducting tissue in the\npulmonary veins or from diseased atrial tissue.\n- Re-entry is more likely to oc
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