Left Ventricular Hypertrophy
Also written as LVH.
Cyanosis with left axis deviation and left ventricular hypertrophy is the signature of tricuspid atresia.
What our notes say
- Cyanosis with left axis deviation and left ventricular hypertrophy is the signature of tricuspid atresia. (Paediatrics end of fourth year special supplementary exam: August 2019, Paper On)
- After another syncopal episode, she is referred to a cardiologist who orders an EKG that shows changes of left ventricular hypertrophy and broad Q waves. (CARDIOVASCULAR PATHOLOGY — Past Paper Questions & Answers)
- Aortic stenosis in the elderly is primarily calcific/degenerative, leading to concentric left ventricular hypertrophy and manifesting clinically as the SAD triad (Syncope, Angina, Dyspnea). (🫀 VALVULAR HEART DISEASE — ULTRA DEEP NOTES)
- The ECG shows left axis deviation and left ventricular hypertrophy. (Paediatrics end of fourth year special supplementary exam: August 2019, Paper On)
- Deep S waves in V1 point to left ventricular hypertrophy. (Paediatrics end of second semester exam (MBPE 4300): 4 October 2022, cardiology,)
- | ECG | Ischaemia, arrhythmia (atrial fibrillation), left ventricular hypertrophy | (Heart failure: which tests and why)
Study material on Left Ventricular Hypertrophy
- CARDIOVASCULAR PATHOLOGY
- Cardiovascular system examination revised 2012
- 🫀 VALVULAR HEART DISEASE — ULTRA DEEP NOTES
- CARDIOVASCULAR PATHOLOGY
Practice questions on Left Ventricular Hypertrophy
A 65-year-old man with aortic stenosis develops left ventricular hypertrophy. This adaptation will eventually fail primarily because:
- Myocytes reach maximum size and cannot enlarge further
- Blood supply cannot keep pace with increased metabolic demands
- Sarcomeres become disorganized with further stress
- Apoptosis of myocytes increases progressively
Answer: B. Cardiac hypertrophy increases metabolic demands of the enlarged myocytes. When blood supply cannot match these demands (relative ischemia), the adaptation fails and progresses to heart failure. While apoptosis may occur later and sarcomere disorganization happens, the primary reason for failure is inadequate perfusion of the hypertrophied muscle mass creating an energy deficit. ---