Non-Cardiogenic Pulmonary Oedema
Also written as NCPO.
| Respiratory | Alveolar‑capillary membrane, surfactant production by type II pneumocytes | Inflammatory cytokines impair surfactant synthesis and increase alveolar permeability, leading to non‑cardiogenic pulmonary oedema (ARDS). |
What our notes say
- | Respiratory | Alveolar‑capillary membrane, surfactant production by type II pneumocytes | Inflammatory cytokines impair surfactant synthesis and increase alveolar permeability, leading to non‑cardiogenic pulmonary oedema (ARDS). | (Sepsis and septic shock)
- *After this note, you should be able to differentiate cardiogenic from non-cardiogenic pulmonary oedema, understand their pathophysiology, recognise clinical features, interpret investigations, and initiate emergency management. (Pulmonary oedema and ARDS)
- | Cardiogenic Pulmonary Oedema (CPO) | Non-Cardiogenic Pulmonary Oedema (NCPO) / ARDS | (Pulmonary oedema and ARDS)
- Fluid Overload: Exercise extreme caution with aggressive fluids in patients with severe malaria (risk of non-cardiogenic pulmonary oedema) and acute renal failure. (Approach to fever)
- | Chest X-ray | Pulmonary oedema, pneumonia. | May show signs of non-cardiogenic pulmonary oedema in severe malaria. | (Malaria, including severe malaria)
- Diffuse alveolar damage → increased capillary permeability → protein-rich exudate floods alveoli (non-cardiogenic pulmonary oedema, distinguishing it from cardiogenic oedema) (Ovarian & Colorectal Cancer: Risks, Pathogenesis, Staging)